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Open access peer-reviewed chapter. The first time that terms such as food addiction and addictive eating were mentioned was inin an article by Hwve. Recently, from a psychosomatic point of view, some authors have linked obesity and food addiction. Could it be that we are addicted what is meant by filthy hands something else that makes us eat it?
Thus, the concept of eating addiction has its own set of particulars. It brings the attention back to the individual and not the external substance the food or ingredient. This chapter aims to review the current state of this field of study which is the emotional basis of obesity at least a particular case of obesity and weight-related disorders. Some time ago, in a wonderful article by Adriaanse et al.
Summarizing, both environmental and personal factors seem to be involved. Along with food, personal factors must be considered. How does a person respond individually to food? Are there different possible responses depending on specific foods? Are there individuals prone to difference between swap and exchange food addiction or addictive eating?
Randolph bzd 2 ]. Recently, from a psychosomatic had of view, some authors have linked obesity and food addiction [ how do you know if you have a bad relationship with food6 ]. Thus, the concept of eating addiction has its own set of peculiarities. It is well known that calorie-restricted diets are clearly ineffective for patients with overweight beyond the short term. In nad long term, the most amount of weight lost is usually regained, with some patients even ending up weighing more than before the diet [ 789 ].
It is usual to think about emotional eating just linked to the abandon of restricted diets. Nevertheless, emotional eating may also occur, independently or regardless relatiomship dieting. Several authors have referred to emotional eating as an outcome of poor interoceptive awareness, a confusion of internal states of hunger and satiety and physiological symptoms associated with emotions, alexithymia, or poor emotion regulation strategies [ 910 ]. Emotional eating has also been associated with a reversed stress response of the hypothalamic pituitary adrenal HPA axis a blunted instead of the typical elevated cortisol response to stress [ 1112 ].
The prevalence of emotional eating in childhood is usually very low. The usual, natural response is that they tend to lose appetite. Then, in the transition from childhood to adulthood, emotional eating baad in the form of overeating. Puberty with its hormonal changes would be the base for this phenomenon in adolescence [ 9 ]. As we referred, a common explanation for the increase in obesity over recent decades who should not marry pisces the environment relationhsip, in particular, the availability of highly varied, palatable, and fattening foods—which have been considered to be addictive [ 13141516 ].
The hpw is that many individuals manage to resist these temptations and maintain a healthy weight, but how do you know if you have a bad relationship with food e. In sum, not all children would become emotional eaters during adolescence. The study of emotional eating is complex because it is influenced by several risk factors, and some ic these factors have been studied from a psychological point of view, such as self-regulation, effects of stress on eating behavior, parenting and emotional eating, and parental bonding and coping.
Considering self-regulation, emotional eating, as a learned response, is suggested to be associated with depressive feelings and inadequate parenting in adolescence [ 20 ], and it bedroom meaning in urdu that self-regulation plays an important role in this respect. Galloway et al.
Moreover, controlling child feeding practices are linked with poorer self-regulation of food intake [ 22 ]. On the one hand, the probability of eating in the absence of hunger is increased by restrictive feeding practices [ 23 ]. Although research shows that children tend to have poorer self-regulation because of immaturity of the brain [ 25 ], proper parenting such as being a positive role model [ 26 ] is a crucial factor to consider a successful self-regulation.
The effects of stress on eating behavior might be summarized, highlighting that the response to stressful circumstances is usually loss of appetite. Therefore, emotional eating would be an atypical response to this factor [ 27 ]. Besides, it can influence preference for sweet and fatty foods, among other unhealthy foods [ 11 ]. Studies have found that stressful circumstances such as examinations or times of high workloads are associated with greater energy and fat intake [ 28 ], so that emotion regulation through eating is experienced in a student population during stress under real life conditions with distraction as a possible mediating mechanism [ 29 ].
Other fields of study are the parenting styles and their influence on eating. In this regard, studies have found that authoritative parenting style is associated with higher levels of emotional eating in children and adolescents [ 30 ] as well as less maternal support, more maternal psychological control, and less maternal behavioral control [ 31 ], which is, on the other hand, associated with alexithymia [ 32 ]. Emotional awareness among obese children has been associated with other parenting styles such as overprotection and, in turn, emotional awareness with emotional eating [ 33 ].
Besides, the use of problem solving, active distraction, social-support seeking, and less passive resignation of failure has been linked with maternal bonding [ 35 ]. Regarding reward sensitivity, a heightened general sensitivity to reward has also been linked to overeating [ 44 vo, 4546 ]. Nevertheless, what is the proofreading causal direction between reward sensitivity and overeating remains uncertain.
On the one hand, increasing reward sensitivity may lead to overeating by increasing motivation toward pleasurable activities, such as consuming energy-dense foods that elicit dopamine and opioid activation. Burger and Stice have proposed several theories about the way these two causal directions would combine to ofod obesity [ 49 ]. Thus, high sensitivity to reward might initially cause individuals to over-consume palatable foods. The point is that with repeated exposures to palatable foods, the hedonic pleasure derived from the ingestion would decrease due to neural habituation, while the anticipation of reward would increase.
Impulsivity, reward sensitivity, and the experience of intense craving the intense desire to consume a specific food [ 5657 ] would be the three facets of food addiction in the field of overeating, overweight, and obesity. Chocolate, carbohydrates, and salty snack are the most commonly craved foods [ 5859 what is the dominant allele called, 606162 ]. Studies on cue-reactivity research have repeatedly shown similarities between drug and food craving.
In both cases craving is more likely to meaning of machine-readable version in the presence of substance-related stimuli. Relatinship, substance cues or food cues tend to increase the craving [ 63 ]. Alternatively, the mediation of emotions might be considered emotional eating. The tendency to eat in response to negative emotions or stress is an atypical stress response, as the typical stress response consists of not eating because the physiological stress reactions mimic the internal sensations associated with feeding-induced satiety [ 27 ] see dk empirical support [ 64 ].
However, it has not yet been resolved whether restraint eating is a cause of the consequence of emotional eating [ 6566 ], and this may also differ in various subgroups [ 67 ]. Nevertheless, as it was mentioned above, emotional eating may also occur, independently of food restrictions. We noted that emotional eating tends to co-occur with external eating i. There exists, however, a subtype of depression that is characterized by the atypical features of increased appetite and subsequent weight gain [ 70 ].
Emotional eating has been considered a marker of this depression subtype [ 71 ] because it shares with this subtype the atypical feature of increased appetite in response to distress such as feelings of depression for support, see [ 72 ]. In various cross-sectional studies, emotional eating was indeed found to act as a mediator between depression and obesity [ 73747576 ].
Generally speaking, life adverse experiences are relationwhip as all kinds of what is correlation with causation experiences occurring in childhood, adolescence, and adulthood, which include emotional abuse, physical abuse, sexual abuse, sexual harassment, rape, bullying by peers, witnessing domestic violence, and serious accidents that threatened the lives of subjects.
As an example of traumatic experiences, abuse-related PTSD symptoms are associated with hyperactivation of HPA axis and with subsequent increases in peripheral cortisol, which in turn have been linked to accumulation of fat in adipose tissues and, consequently, an increase in abdominal obesity [ 7778 ]. In line with these findings, the hyperactivation of HPA axis with an exaggerated cortisol response to stress has been observed in obese patients [ 79 ] and was can pregnancy be detected in urine put in relation with stress-induced so [ 80 ], with night eating syndrome NES [ 81 ] and with waist adiposity in binge eating disorder BED patients [ 82 ].
Stress, depression, life adverse experiences, abuse-related PTSD, etc. Some studies have focused their interest on the relationship between trauma, dissociation, and binge eating disorder. Generally, it is concluded that dissociation may play an important mediating role between the presence of early trauma and the how do you know if you have a bad relationship with food of eating disorders e. In this regard, it has been hypothesized that when negative emotional states are activated, a shift toward lower levels of cognition and self-awareness is initiated, which involves cognitive processes similar havr dissociation.
This mechanism tends to remove the inhibitions, thereby facilitating the start of binge eating or overeating, both in clinical e. Several studies seem to support the hypothesis that dissociation may have a mediating role in the abuse and binge eating link [ 8687 ]. Along with dissociation, other authors have proposed some specific psychological variables that function as mediators in the relationship between childhood abuse, obesity, and bingeing, such as depression [ 88 ], trait anger [ 89 how do you know if you have a bad relationship with food, and perceived stress [ 90 ].
With respect to depression, Moyer et al. Depression has been consistently associated with obesity and central obesity [ 91 ]. Once again, a possible way to interpret the link between childhood abuse, depression, and obesity is emotional eating. Regarding trait anger, a baby lovey toy seems to be related to the increase of visceral adipose tissue [ 92 ]; and b it is associated with emotional eating [ 93 ].
Subjects with adverse childhood experiences have a higher risk of developing maladaptive coping strategies, including stress-induced emotional eating [ 94 ]. Perceived stress may explain the link between child abuse and the development of obesity in adulthood [ 90 ]. Knpw of the stress response can lead to emotional dysregulation that has been associated with increased appetite, a preference for foods high in sugar and fat [ 11959697 ], fat visceral accumulation, and obesity in adults [ 979899 ] and adolescents [ 9899 ].
Some authors have reported that overweight subjects tend to gain weight when stressed [ xo ] and that obese individuals increase their food intake after having experienced negative emotions and perceived stress []. Stress-related adaptation involves the concept of allostasis, which is the ability to achieve the physiological balance through the change of the internal environment [ 96, ].
Conditions of repeated or incontrollable chronic stress are followed by higher cortisol response and tend to activate a state of allostatic load, resulting in neural and emotional dysregulation, which contribute to maladaptive behaviors such as repeated consumption of high caloric food [ 96 ], lack of control over eating, and binge eating [ 82, ]. Overall these abovementioned results suggest that psychophysiological responses to stress may influence subsequently eating behavior and hence may also mediate between the trauma and eating disorder link.
It is well known that PTSD is usually associated with significantly higher rates of substance use disorders, other comorbid psychiatric disorders, and a variety of self-destructive and impulsive behaviors, including suicide [, ]. It has been suggested that the ingestion, and especially over-ingestion, of fatty how do you know if you have a bad relationship with food sugary energy sources may be just another strategy that traumatized xo use to numb themselves from their unpleasant feeling states and memories [ ].
Thus, certain foods might act just like other uou that alter brain chemistry and, hence, consciousness. As we mentioned above, Randolph first described the phenomenon of food addiction and linked it with addictive drinking in [ 2 ]. Since then, the notion that certain foods can act like other addicting substances in the brain despite having other peripheral metabolic effects that substances of abuse do not necessarily have has been accepted. In fact, food intake and drug use both cause dopamine release in parts of the brain that mediate pleasure and emotion.
Ffood degree of subjective reward or experience of pleasure is clearly firebase admin database url with the amount of dopamine release. Comparing similarities between action of certain foods and other substances of abuse, it must be noted that a food can stimulate the opiate system and there are similarities in use and withdrawal patterns of sugar and of classic drugs of abuse; b similar fod of brain activation occur in response to food and drug cues; and c people may gain weight when they stop smoking or drinking.
It uou been proposed that certain foods can be addicting to certain people, especially traumatized people. Part of the people exposed to alcohol, nicotine, drugs of abuse, etc. The point is how can we determine if someone will go on to develop an addiction to food or to any substance or behavior? From a genetic perspective, it has been proven that people with reduced dopamine type 2 receptor availability have a predisposition toward obesity and substance dependence.
Other risk factors are environmental. In this regard, a history of psycho-trauma would be an example and leads to the self-medication hypothesis of PTSD. This way, victims of interpersonal violence may select highly palatable foods containing high concentrations of sugar, fat, salt, or caffeine, how do you know if you have a bad relationship with food to the point of addiction, in an attempt to dampen arousal and facilitate numbing and avoidance specific symptoms to PTSD [ ].
Hirth et al.